NEFIRACETAM is a pyrrolidone to the psychotropic drug, and its structure is similar to that of acetaminophen, namely piracetam. Structurally, it is more different from aniracetam or oxiracetam (very similar to piracetam) but retains some common characteristics with them.
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The mechanism of action is two so-called ways of NEFIRACETAM. The first way is to prolong the opening of calcium channels (binding to PKA and GI / O proteins) that enhance receptor response, regardless of synapses; The second pathway is associated with PKC and CaMKII, which then completes the response due to cholinergic receptors (which are then released by the most exciting neurotransmitters at the presynaptic level, such as nicotine). The previous pathway (calcium channel) is important in the long term, while the latter pathway (PKC / CaMKII) has an impact on the enhancement of neural signals and their transmission. Some other minor ways can be partial agonists of NDMA receptors bound to glycine (which can enhance the interaction of certain levels of glycine, thereby weakening the interaction), and increase their communication with the ligand acetylcholine, musk acetylcholine receptor. Nefiracetam is a cognitive enhancer, which prolongs the role of calcium in activated neurons and improves memory function by increasing the exposure of acetylcholine and glutamate in synapses.
NEFIRACETAM is a cognitive enhancer (seminal drug) of the racetam family. It was originally derived from the parent molecule piracetam, but it is most similar to aniracetam in structure. Because NEFIRACETAM and aniracetam are fat-soluble drugs. They are used to improve the treatment of memory and cognitive impairment. For single-use, NEFIRACETAM had no significant effect on the quality of memory. Nefexitan can affect the memory formation of daily use for at least 7 days, which has repeatedly proved that it has the same effect in animal studies and in humans. Long-term use is also associated with a higher degree of neurogenesis, which does not occur on a single reception.
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