Abamectin Powder CAS 71751-41-2

AverMectin is a class of sixteen membered macrolide compounds with insecticidal, acaricidal, and nematode killing activities, produced by fermentation of Streptomyces avermitilis, a gray Streptomyces species. Natural Abamectin Powder contains 8 components, mainly consisting of 4 types: A1a, A2a, B1a, and B2a, with a total content of ≥ 80%; The corresponding four smaller proportion homologues are A1b, A2b, B1b, and B2b, with a total content of ≤ 20%. At present, the finished products on the market are mainly composed of AverMectin B1a-AverMectin B1b Mixt as the main insecticidal ingredient, calibrated based on the content of B1a.

What is the poisoning mechanism of Abamectin?

Avermectin powder can increase the release of the neurotransmitter γ-amino acid, slightly and temporarily inhibit the central nervous system (the damage of the central nervous system is the most common) and gastrointestinal symptoms. When a large amount of Abamectin powder is absorbed into the blood and enters the center through the blood-brain barrier, the functions of the cerebral cortex, Purkinje cells in the cerebellum cortex and striatum-substantia nigra fibers in the central nervous system will be widely inhibited, resulting in anxiety, irritability, drowsiness, depression, convulsion and coma. In severe cases, coma, respiratory failure and shock may occur, even central depression and respiratory depression (central respiratory failure).If it is not relieved for a long time, it will die because of frequent convulsions, suffocation or ventricular fibrillation.

At present, avermectin has no special antidote. It may have an effect by inhibiting the pharmacological action of GABAA receptor, but it lacks clinical practice. Therefore, the key to successful rescue is to remove poisons as soon as possible, prevent the absorption of poisons and promote the excretion of poisons.

What pest control effect of avermectin is the best?

Abamectin Powder has always been favored by growers for its excellent cost-effectiveness. At present, it is a more mainstream pesticide on the market. Its mechanism of action is different from that of general insecticides in that it interferes with neurophysiological activities, stimulates the release of r-aminobutyric acid, and r-aminobutyric acid has an inhibitory effect on nerve conduction in arthropods. Mites, nymphs, insects, and larvae will experience paralysis symptoms upon contact with the pesticide, stop moving and feeding, and die 2-4 days later. But it cannot kill the insect eggs.
Abamectin is not only an insecticide, but also a acaricide and can be used to kill nematodes. It has the advantages of contact killing, stomach toxicity, and strong penetration. It is a macrocyclic lactone disaccharide compound. It is a natural product isolated from soil microorganisms, which has contact killing and stomach poisoning effects on insects and mites, and has a weak fumigation effect, but no systemic effect. But it has a strong penetrating effect on the leaves, can kill pests under the epidermis, and has a long residual period. Because it does not cause rapid dehydration of insects, its lethal effect is slower. However, although it has a direct killing effect on predatory and parasitic natural enemies, the damage to beneficial insects is minimal due to the low residue on the plant surface. It has a significant effect on root knot nematodes. However, due to the high frequency of use of this product and the emergence of resistance, we generally do not recommend using AverMectin B1 alone to control pests. It is usually used in combination with other pesticides. It is recommended that when using avermectin, attention should be paid to rotating the use of drugs to delay the development of resistance.

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